[1]
|
Sang, W., Chen, S., Lin, L., et al. (2022) Antioxidant Mitoquinone Ameliorates EtOH-LPS Induced Lung Injury by In-hibiting Mitophagy and NLRP3 Inflammasome Activation. Frontiers in Immunology, 13, Article 973108.
https://doi.org/10.3389/fimmu.2022.973108
|
[2]
|
Siggins, R.W., Mcternan, P.M., Simon, L., et al. (2023) Mito-chondrial Dysfunction: At the Nexus between Alcohol-Associated Immunometabolic Dysregulation and Tissue Injury. International Journal of Molecular Sciences, 24, Article 8650. https://doi.org/10.3390/ijms24108650
|
[3]
|
Bellar, A., Welch, N., Dasarathy, J., et al. (2023) Peripheral Blood Mononuclear Cell Mitochondrial Dysfunction in Acute Al-cohol-Associated Hepatitis. Clinical and Translational Medicine, 13, e1276.
https://doi.org/10.1002/ctm2.1276
|
[4]
|
Kołota, A., Głąbska, D., Oczkowski, M. and Gromadzka-Ostrowska, J. (2020) Oxidative Stress Parameters in the Liver of Growing Male Rats Receiving Various Alcoholic Beverages. Nutri-ents, 12, Article 158.
https://doi.org/10.3390/nu12010158
|
[5]
|
邱鲲羽, 黄延焱. 酒精性脑损伤作用机制的研究进展[J]. 老年医学与保健, 2022, 28(5): 1160-1163.
|
[6]
|
Sivolap, Y.P. (2021) [Treatment of Alcohol Withdrawal Syndrome]. Zhurnal nevrologii i psikhiatrii imeni SS Korsakova, 121, 139-144. https://doi.org/10.17116/jnevro2021121061139
|
[7]
|
Laniepce, A., Cabé, N., André, C., et al. (2020) The Effect of Alcohol Withdrawal Syndrome Severity on Sleep, Brain and Cognition. Brain Communications, 2, fcaa123. https://doi.org/10.1093/braincomms/fcaa123
|
[8]
|
Hansen, K.B., Yi, F., Perszyk, R.E., Menniti, F.S. and Traynelis, S.F. (2017) NMDA Receptors in the Central Nervous System. In: Burnashev, N. and Szepetowski, P., Eds., NMDA Re-ceptors, Humana Press, New York, 1-80.
https://doi.org/10.1007/978-1-4939-7321-7_1
|
[9]
|
Barkley-Levenson, A.M., Lee, A. and Palmer, A.A. (2021) Genetic and Pharmacological Manipulations of Glyoxalase 1 Mediate Ethanol Withdrawal Seizure Susceptibility in Mice. Brain Sciences, 11, Article 127.
https://doi.org/10.3390/brainsci11010127
|
[10]
|
Jung, M.E. and Metzger, D.B. (2010) Alcohol Withdrawal and Brain Injuries: Beyond Classical Mechanisms. Molecules, 15, 4984-5011. https://doi.org/10.3390/molecules15074984
|
[11]
|
Jung, M.E., Yan, L.J., Forster, M.J. and Simpkins, J.W. (2008) Ethanol Withdrawal Provokes Mitochondrial Injury in an Estrogen Preventable Manner. Journal of Bioenergetics and Biomembranes, 40, 35-44.
https://doi.org/10.1007/s10863-008-9129-y
|
[12]
|
Jung, M.E., Ju, X., Metzger, D.B. and Simpkins, J.W. (2012) Ethanol Withdrawal Hastens the Aging of Cytochrome c Oxidase. Neurobiology of Aging, 33, 618.e21-618.e32. https://doi.org/10.1016/j.neurobiolaging.2011.02.002
|
[13]
|
Chatterjee, O., Gopalakrishnan, L., Pullimamidi, D., et al. (2022) A Molecular Network Map of Orexin-Orexin Receptor Signaling System. Journal of Cell Communication and Signaling, 17, 217-227.
https://doi.org/10.1007/s12079-022-00700-3
|
[14]
|
Nambu, T., Sakurai, T., Mizukami, K., et al. (1999) Distribution of Orexin Neurons in the Adult Rat Brain. Brain Research, 827, 243-260. https://doi.org/10.1016/S0006-8993(99)01336-0
|
[15]
|
Bayerlein, K., Kraus, T., Leinonen, I., et al. (2011) Orexin A Expression and Promoter Methylation in Patients with Alcohol Dependence Comparing Acute and Protracted Withdrawal. Alcohol, 45, 541-547.
https://doi.org/10.1016/j.alcohol.2011.02.306
|
[16]
|
Lawrence, A.J., Cowen, M.S., Yang, H.J., et al. (2006) The Orexin System Regulates Alcohol-Seeking in Rats. British Journal of Pharmacology, 148, 752-759. https://doi.org/10.1038/sj.bjp.0706789
|
[17]
|
Lei, K., Wegner, S.A., Yu, J.H. and Hopf, F.W. (2016) Orexin-1 Re-ceptor Blockade Suppresses Compulsive-Like Alcohol Drinking in Mice. Neuropharmacology, 110, 431-437. https://doi.org/10.1016/j.neuropharm.2016.08.008
|
[18]
|
Michalak, A., Lach, T. and Cichoż-Lach, H. (2021) Oxida-tive Stress—A Key Player in the Course of Alcohol-Related Liver Disease. Journal of Clinical Medicine, 10, Article 3011. https://doi.org/10.3390/jcm10143011
|
[19]
|
Gonzaga, N.A., Do Vale, G.T., Parente, J.M., et al. (2018) Etha-nol Withdrawal Increases Blood Pressure and Vascular Oxidative Stress: A Role for Angiotensin Type 1 Receptors. Journal of the American Society of Hypertension, 12, 561-573. https://doi.org/10.1016/j.jash.2018.03.012
|
[20]
|
Huang, M.C., Chen, C.H., Peng, F.C., et al. (2009) Alterations in Oxidative Stress Status during Early Alcohol Withdrawal in Alcoholic Patients. Journal of the Formosan Medical Asso-ciation, 108, 560-569.
https://doi.org/10.1016/S0929-6646(09)60374-0
|
[21]
|
Mohebbi, E., Molavi, M., Mohammadzadeh, M., et al. (2020) Clavulanic Acid Improves Ethanol Withdrawal Symptoms in Rats. Iranian Journal of Basic Medical Sciences, 23, 730-736.
|
[22]
|
Sordi, A.O., Pechansky, F., Kessler, F.H., et al. (2014) Oxidative Stress and BDNF as Possible Markers for the Severity of Crack Cocaine Use in Early Withdrawal. Psychopharmacology, 231, 4031-4039.
https://doi.org/10.1007/s00213-014-3542-1
|
[23]
|
Parthasarathy, R., Kattimani, S. and Sridhar, M.G. (2015) Oxida-tive Stress during Alcohol Withdrawal and Its Relationship with Withdrawal Severity. Indian Journal of Psychological Medicine, 37, 175-180.
https://doi.org/10.4103/0253-7176.155617
|
[24]
|
Rauniyar, N., Stevens, S.M., Prokai-Tatrai, K. and Prokai, L. (2009) Characterization of 4-Hydroxy-2-Nonenal-Modified Peptides by Liquid Chromatography-Tandem Mass Spec-trometry Using Data-Dependent Acquisition: Neutral Loss-Driven MS3 versus Neutral Loss-Driven Electron Capture Dissociation. Analytical Chemistry, 81, 782-789.
https://doi.org/10.1021/ac802015m
|
[25]
|
Prokai, L., Yan, L.J., Vera-Serrano, J.L., et al. (2007) Mass Spectrome-try-Based Survey of Age-Associated Protein Carbonylation in Rat Brain Mitochondria. Journal of Mass Spectrometry, 42, 1583-1589.
https://doi.org/10.1002/jms.1345
|
[26]
|
Adachi, J. (2000) [Membrane Disorder and Free Radical]. Nihon Hoigaku Zasshi, 54, 356-360.
|
[27]
|
Lehotsky, J., Kaplan, P., Matejovicova, M., et al. (2002) Ion Transport Systems as Targets of Free Radicals during Ischemia Reperfusion Injury. General Physiology and Biophysics, 21, 31-37.
|
[28]
|
Shulman, R.G., Rothman, D.L., Behar, K.L. and Hyder, F. (2004) Energetic Basis of Brain Activity: Implications for Neuroimaging. Trends in Neurosciences, 27, 489-495. https://doi.org/10.1016/j.tins.2004.06.005
|
[29]
|
Ishihara, Y., Takemoto, T., Ishida, A. and Yamazaki, T. (2015) Protective Actions of 17β-Estradiol and Progesterone on Oxidative Neuronal Injury Induced by Organometallic Compounds. Oxidative Medicine and Cellular Longevity, 2015, Article ID: 343706. https://doi.org/10.1155/2015/343706
|
[30]
|
Khan, M., Shah, S.A. and Kim, M.O. (2018) 17β-Estradiol via SIRT1/Acetyl-p53/NF-kB Signaling Pathway Rescued Postnatal Rat Brain against Acute Ethanol Intoxication. Molecular Neurobiology, 55, 3067-3078.
https://doi.org/10.1007/s12035-017-0520-8
|
[31]
|
Sagara, Y. (1998) Induction of Reactive Oxygen Species in Neu-rons by Haloperidol. Journal of Neurochemistry, 71, 1002-1012. https://doi.org/10.1046/j.1471-4159.1998.71031002.x
|
[32]
|
Tsialtas, I., Georgantopoulos, A., Karipidou, M.E., et al. (2021) Anti-Apoptotic and Antioxidant Activities of the Mitochondrial Estrogen Receptor Beta in N2A Neuroblastoma Cells. International Journal of Molecular Sciences, 22, Article 7620. https://doi.org/10.3390/ijms22147620
|
[33]
|
Ramezani, A., Goudarzi, I., Lashkarbolouki, T., et al. (2011) Neuro-protective Effects of the 17β-Estradiol against Ethanol-Induced Neurotoxicity and Oxidative Stress in the Developing Male Rat Cerebellum: Biochemical, Histological and Behavioral Changes. Pharmacology Biochemistry and Behavior, 100, 144-151.
https://doi.org/10.1016/j.pbb.2011.07.010
|
[34]
|
Stenger, C., Naves, T., Verdier, M., et al. (2011) The Cell Death Response to the ROS Inducer, Cobalt Chloride, in Neuroblastoma Cell Lines According to p53 Status. International Journal of Oncology, 39, 601-609.
|
[35]
|
Sokołowska, P., Urbańska, A., Biegańska, K., et al. (2014) Orexins Protect Neuronal Cell Cultures against Hypoxic Stress: An Involvement of Akt Signaling. Journal of Molecular Neuroscience, 52, 48-55.
https://doi.org/10.1007/s12031-013-0165-7
|
[36]
|
Fernandez-Rodriguez, S., Cano-Cebrian, M.J., Rius-Perez, S., et al. (2022) Different Brain Oxidative and Neuroinflammation Status in Rats during Prolonged Abstinence Depending on Their Ethanol Relapse-Like Drinking Behavior: Effects of Ethanol Reintroduction. Drug and Alcohol Dependence, 232, Article ID: 109284.
https://doi.org/10.1016/j.drugalcdep.2022.109284
|
[37]
|
Yen, C.H., Ho, P.S., Yeh, Y.W., et al. (2017) Differential Cytokine Levels between Early Withdrawal and Remission States in Patients with Alcohol Dependence. Psychoneuroen-docrinology, 76, 183-191.
https://doi.org/10.1016/j.psyneuen.2016.10.015
|
[38]
|
Leon, B.E., Kang, S., Franca-Solomon, G., et al. (2021) Al-cohol-Induced Neuroinflammatory Response and Mitochondrial Dysfunction on Aging and Alzheimer’s Disease. Fron-tiers in Behavioral Neuroscience, 15, Article 778456.
https://doi.org/10.3389/fnbeh.2021.778456
|
[39]
|
Girard, M., Malauzat, D. and Nubukpo, P. (2019) Serum Inflam-matory Molecules and Markers of Neuronal Damage in Alcohol-Dependent Subjects after Withdrawal. The World Jour-nal of Biological Psychiatry, 20, 76-90.
https://doi.org/10.3389/fnbeh.2021.778456
|
[40]
|
Messal, N., Fernandez, N., Dayot, S., et al. (2018) Ectopic Ex-pression of OX1R in Ulcerative Colitis Mediates Anti-Inflammatory Effect of Orexin-A. Biochimica et Biophysica Acta (BBA)—Molecular Basis of Disease, 1864, 3618-3628. https://doi.org/10.1016/j.bbadis.2018.08.023
|
[41]
|
Hu, S., Niu, J., Zhang, R., et al. (2020) Orexin A Associates with Inflammation by Interacting with OX1R/OX2R Receptor and Activating Prepro-Orexin in Cancer Tissues of Gastric Cancer Patients. Gastroenterología y Hepatología, 43, 240-247. https://doi.org/10.1016/j.gastrohep.2019.10.006
|
[42]
|
Xiong, X., White, R.E., Xu, L., et al. (2013) Mitigation of Murine Focal Cerebral Ischemia by the Hypocretin/Orexin System Is Associated with Reduced Inflammation. Stroke, 44, 764-770.
https://doi.org/10.1161/STROKEAHA.112.681700
|
[43]
|
Nakamachi, T., Endo, S., Ohtaki, H., et al. (2005) Orex-in-1 Receptor Expression after Global Ischemia in Mice. Regulatory Peptides, 126, 49-54. https://doi.org/10.1016/j.regpep.2004.08.037
|
[44]
|
Tunisi, L., Forte, N., Fernández-Rilo, A.C., et al. (2019) Orex-in-A Prevents Lipopolysaccharide-Induced Neuroinflammation at the Level of the Intestinal Barrier. Frontiers in Endo-crinology, 10, Article 219.
https://doi.org/10.3389/fendo.2019.00219
|
[45]
|
Gorin, M.A. and Pan, Q. (2009) Protein Kinase C Epsilon: An Oncogene and Emerging Tumor Biomarker. Molecular Cancer, 8, Article No. 9. https://doi.org/10.1186/1476-4598-8-9
|
[46]
|
Kang, J.H., Toita, R., Kim, C.W. and Katayama, Y. (2012) Protein Ki-nase C (PKC) Isozyme-Specific Substrates and Their Design. Biotechnology Advances, 30, 1662-1672. https://doi.org/10.1016/j.biotechadv.2012.07.004
|
[47]
|
Zhou, H.Z., Karliner, J.S. and Gray, M.O. (2002) Moderate Alcohol Consumption Induces Sustained Cardiac Protection by Activating PKC-Epsilon and Akt. American Journal of Physiology-Heart and Circulatory Physiology, 283, H165-H174. https://doi.org/10.1152/ajpheart.00408.2001
|
[48]
|
Dilly, G.A., Kittleman, C.W., Kerr, T.M., et al. (2022) Cell-Type Specific Changes in PKC-Delta Neurons of the Central Amygdala during Alcohol Withdrawal. Translational Psychiatry, 12, Article No. 289.
https://doi.org/10.1038/s41398-022-02063-0
|
[49]
|
Hernández-Rojas, R., Jiménez-Arellano, C., De La Fuente-Granada, M., et al. (2022) The Interplay between Estrogen Receptor β and Protein Kinase C, a Crucial Collabora-tion for Medulloblastoma Cell Proliferation and Invasion. Cellular Signalling, 92, Article ID: 110246. https://doi.org/10.1016/j.cellsig.2022.110246
|
[50]
|
Chen, J., He, Y., Wu, Y., et al. (2018) Single Ethanol With-drawal Regulates Extrasynaptic δ-GABAA Receptors via PKCδ Activation. Frontiers in Molecular Neuroscience, 11, Article 141. https://doi.org/10.3389/fnmol.2018.00141
|
[51]
|
Bohnsack, J.P., Hughes, B.A., O’buckley, T.K., et al. (2018) Histone Deacetylases Mediate GABAA Receptor Expression, Physiology, and Behavioral Maladaptations in Rat Models of Alcohol Dependence. Neuropsychopharmacology, 43, 1518-1529. https://doi.org/10.1038/s41386-018-0034-8
|
[52]
|
Jung, M.E., Metzger, D.B. and Das, H.K. (2016) The Role of Presenilin-1 in the Excitotoxicity of Ethanol Withdrawal. Journal of Pharmacology and Experimental Therapeutics, 358, 516-526. https://doi.org/10.1124/jpet.116.233361
|
[53]
|
Ku, B.M., Lee, Y.K., Jeong, J.Y., et al. (2007) Etha-nol-Induced Oxidative Stress Is Mediated by p38 MAPK Pathway in Mouse Hippocampal Cells. Neuroscience Letters, 419, 64-67. https://doi.org/10.1016/j.neulet.2007.03.049
|
[54]
|
Valles, S.L., Borras, C., Gambini, J., et al. (2008) Oestradiol or Genistein Rescues Neurons from Amyloid β-Induced Cell Death by Inhibiting Activation of p38. Aging Cell, 7, 112-118. https://doi.org/10.1111/j.1474-9726.2007.00356.x
|
[55]
|
Stuart, R. (2002) Insertion of Proteins in-to the Inner Membrane of Mitochondria: The Role of the Oxa1 Complex. Biochimica et Biophysica Acta (BBA)—Molecular Cell Research, 1592, 79-87.
https://doi.org/10.1016/S0167-4889(02)00266-5
|
[56]
|
Jung, M.E. (2015) Alcohol Withdrawal and Cerebellar Mi-tochondria. Cerebellum, 14, 421-437.
https://doi.org/10.1007/s12311-014-0598-8
|
[57]
|
Johnson, J., Mercado-Ayon, E., Mercado-Ayon, Y., et al. (2021) Mitochondrial Dysfunction in the Development and Progression of Neurodegenerative Diseases. Archives of Biochemis-try and Biophysics, 702, Article ID: 108698.
https://doi.org/10.1016/j.abb.2020.108698
|
[58]
|
Jung, M.E., Wilson, A.M., Ju, X., et al. (2009) Ethanol Withdrawal Provokes Opening of the Mitochondrial Membrane Permeability Transition Pore in an Estrogen-Preventable Manner. Journal of Pharmacology and Experimental Therapeutics, 328, 692-698. https://doi.org/10.1124/jpet.108.146829
|
[59]
|
Jung, M.E. and Metzger, D.B. (2015) Aberrant Histone Acetylation Promotes Mitochondrial Respiratory Suppression in the Brain of Alcoholic Rats. Journal of Pharmacology and Experi-mental Therapeutics, 352, 258-266.
https://doi.org/10.1124/jpet.114.219311
|
[60]
|
Gaignard, P., Frechou, M., Liere, P., et al. (2018) Sex Differences in Brain Mitochondrial Metabolism: Influence of Endogenous Steroids and Stroke. Journal of Neuroendocrinology, 30, e12497. https://doi.org/10.1111/jne.12497
|
[61]
|
Chen, J.Q. and Yager, J.D. (2004) Estrogen’s Effects on Mito-chondrial Gene Expression: Mechanisms and Potential Contributions to Estrogen Carcinogenesis. Annals of the New York Academy of Sciences, 1028, 258-272.
https://doi.org/10.1196/annals.1322.030
|
[62]
|
Rettberg, J.R., Yao, J. and Brinton, R.D. (2014) Estrogen: A Master Regulator of Bioenergetic Systems in the Brain and Body. Frontiers in Neuroendocrinology, 35, 8-30. https://doi.org/10.1016/j.yfrne.2013.08.001
|
[63]
|
Klinge, C.M. (2017) Estrogens Regulate Life and Death in Mito-chondria. Journal of Bioenergetics and Biomembranes, 49, 307-324. https://doi.org/10.1007/s10863-017-9704-1
|
[64]
|
Li, M., Meng, Y., Chu, B., et al. (2020) Orexin-A Aggravates Cytotoxicity and Mitochondrial Impairment in SH-SY5Y Cells Transfected with APPswe via p38 MAPK Pathway. An-nals of Translational Medicine, 8, Article 5.
https://doi.org/10.21037/atm.2019.11.68
|