[1]
|
岑海鹏, 宫赫, 李晨晨, 等. 多尺度分析骨质疏松大鼠骨微结构变化[J]. 医用生物力学, 2023, 38(3): 514-520.
|
[2]
|
章振林, 金小岚, 夏维波. 原发性骨质疏松症诊疗指南(2017版)要点解读[J]. 中华骨质疏松和骨矿盐疾病杂志, 2017, 10(5): 411-412.
|
[3]
|
Cole, Z.A., Dennison, E.M. and Cooper, C. (2008) Osteoporosis Epidemiology Update. Current Rheumatology Reports, 10, 92-96. https://doi.org/10.1007/s11926-008-0017-6
|
[4]
|
中国骨质疏松症流行病学调查及“健康骨骼”专项行动结果发布[J]. 中华骨质疏松和骨矿盐疾病杂志, 2019, 12(4): 317-318.
|
[5]
|
Huybrechts, Y., Mortier, G., Boudin, E., et al. (2020) WNT Signaling and Bone: Lessons from Skeletal Dysplasias and Disorders. Frontiers in Endocrinology (Lausanne), 11, Article No. 165. https://doi.org/10.3389/fendo.2020.00165
|
[6]
|
Ghosh, N., Hossain, U., Mandal, A., et al. (2019) The Wnt Signal-ing Pathway: A Potential Therapeutic Target against Cancer. Annals of the New York Academy of Sciences, 1443, 54-74. https://doi.org/10.1111/nyas.14027
|
[7]
|
Hayat, R., Manzoor, M. and Hussain, A. (2022) Wnt Signaling Pathway: A Comprehensive Review. Cell Biology International, 46, 863-877. https://doi.org/10.1002/cbin.11797
|
[8]
|
Bänziger, C., Soldini, D., Schütt, C., et al. (2006) Wntless, a Conserved Membrane Protein Dedicated to the Secretion of Wnt Proteins from Signaling Cells. Cell, 125, 509-522. https://doi.org/10.1016/j.cell.2006.02.049
|
[9]
|
Finkbeiner, M.G., Sawan, C., Ouzounova, M., et al. (2008) HAT Cofactor TRRAP Mediates Beta-Catenin Ubiquitination on the Chromatin and the Regulation of the Canonical Wnt Pathway. Cell Cycle, 7, 3908-3914.
https://doi.org/10.4161/cc.7.24.7354
|
[10]
|
Wu, C. and Nusse, R. (2002) Ligand Receptor Interactions in the Wnt Signaling Pathway in Drosophila. Journal of Biological Chemistry, 277, 41762-41769. https://doi.org/10.1074/jbc.M207850200
|
[11]
|
He, X., Saint-Jeannet, J., Wang, Y., et al. (1997) A Member of the Frizzled Protein Family Mediating Axis Induction by Wnt-5A. Science, 275, 1652-1654. https://doi.org/10.1126/science.275.5306.1652
|
[12]
|
Krasnow, R.E., Wong, L.L. and Adler, P.N. (1995) Dishev-elled Is a Component of the Frizzled Signaling Pathway in Drosophila. Development (Cambridge), 121, 4095-4102. https://doi.org/10.1242/dev.121.12.4095
|
[13]
|
Cselenyi, C.S., Jernigan, K.K., Tahinci, E., et al. (2008) LRP6 Transduces a Canonical Wnt Signal Independently of Axin Degradation by Inhibiting GSK3’s Phosphorylation of β-Catenin. Proceedings of the National Academy of Sciences, 105, 8032-80377. https://doi.org/10.1073/pnas.0803025105
|
[14]
|
Salomon, D., Sacco, P.A., Roy, S.G., et al. (1997) Regulation of β-Catenin Levels and Localization by Overexpression of Plakoglobin and Inhibition of the Ubiquitin-Proteasome System. The Journal of Cell Biology, 139, 1325-1335.
https://doi.org/10.1083/jcb.139.5.1325
|
[15]
|
Wodarz, A. and Nusse, R. (1998) Mechanisms of Writ Signaling in Development. Annual Review of Cell and Developmental Biology, 14, 59-88. https://doi.org/10.1146/annurev.cellbio.14.1.59
|
[16]
|
Shimizu, T., Kagawa, T., Inoue, T., et al. (2008) Stabilized β-Catenin Functions through TCF/LEF Proteins and the Notch/RBP-Jκ Complex To Promote Proliferation and Suppress Differentiation of Neural Precursor Cells. Molecular and Cellular Biology, 28, 7427-7441. https://doi.org/10.1128/MCB.01962-07
|
[17]
|
Vuong, L.T. and Mlodzik, M. (2022) Different Strategies by Distinct Wnt-Signaling Pathways in Activating a Nuclear Transcriptional Response. Current Topics in Developmental Biology, 149, 59-89.
https://doi.org/10.1016/bs.ctdb.2022.02.008
|
[18]
|
Yang, R.B., Lin, F.F., Yang, J., et al. (2022) Retraction Note: Overexpression of CAV3 Facilitates Bone Formation via the Wnt Signaling Pathway in Osteoporotic Rats. Endocrine, 76, 751. https://doi.org/10.1007/s12020-022-03032-9
|
[19]
|
Chagay, N.B., Khayt, G.Y., Vdovina, T.M., et al. (2021) Cystic Fibrosis Being a Polyendocrine Disease (Review). Problems of Endocrinology (Mosk), 67, 28-39. https://doi.org/10.14341/probl12694
|
[20]
|
Liu, J., Xiao, Q., Xiao, J., et al. (2022) Wnt/β-Catenin Signalling: Func-tion, Biological Mechanisms, and Therapeutic Opportunities. Signal Transduction and Targeted Therapy, 7, Article No. 3.
https://doi.org/10.1038/s41392-021-00762-6
|
[21]
|
Amjadi-Moheb, F. and Akhavan-Niaki, H. (2019) Wnt Signaling Pathway in Osteoporosis: Epigenetic Regulation, Interaction with Other Signaling Pathways, and Therapeutic Promises. Journal of Cellular Physiology, 234, 14641-14650.
https://doi.org/10.1002/jcp.28207
|
[22]
|
Teufel, S. and Hartmann, C. (2019) Wnt-Signaling in Skeletal Development. Current Topics in Developmental Biology, 133, 235-279. https://doi.org/10.1016/bs.ctdb.2018.11.010
|
[23]
|
代光明, 任磊, 陈虹, 等. 下调骨细胞TGF-β/Smad4信号可抑制小鼠BMSCs成骨及破骨分化[J]. 基础医学与临床, 2017, 37(6): 786-791.
|
[24]
|
Zhang, X., Li, H., Chen, F., et al. (2021) Icariin Regulates miR-23a-3p-Mediated Osteogenic Dif-ferentiation of BMSCs via BMP-2/Smad5/Runx2 and WNT/β-Catenin Pathways in Osteonecrosis of the Femoral Head. Saudi Pharmaceutical Journal, 29, 1405-1415. https://doi.org/10.1016/j.jsps.2021.10.009
|
[25]
|
Visweswaran, M., Pohl, S., Arfuso, F., et al. (2015) Multi-Lineage Differentiation of Mesenchymal Stem Cells—To Wnt, or Not Wnt. The International Journal of Biochemistry & Cell Biology, 68, 139-147.
https://doi.org/10.1016/j.biocel.2015.09.008
|
[26]
|
Pan, F., Shao, J., Shi, C., et al. (2021) Apigenin Promotes Oste-ogenic Differentiation of Mesenchymal Stem Cells and Accelerates Bone Fracture Healing via Activating Wnt/β-Catenin Signaling. American Journal of Physiology: Endocrinology and Metabolism, 320, E760-E771. https://doi.org/10.1152/ajpendo.00543.2019
|
[27]
|
Liang, Y., Liu, X., Zhou, R., et al. (2021) Chaetocin Promotes Osteogenic Differentiation via Modulating Wnt/Beta- Catenin Signaling in Mesenchymal Stem Cells. Stem Cells Interna-tional, 2021, Article ID: 8888416.
https://doi.org/10.1155/2021/8888416
|
[28]
|
Lademann, F., Tsourdi, E., Hofbauer, L.C., et al. (2020) Thyroid Hor-mone Actions and Bone Remodeling—The Role of the Wnt Signaling Pathway. Experimental and Clinical Endocrinolo-gy & Diabetes, 128, 450-454.
https://doi.org/10.1055/a-1088-1215
|
[29]
|
Zhang, H., Wang, J., Deng, F., et al. (2015) Canonical Wnt Signaling Acts Synergistically on BMP9-Induced Osteo/Odontoblastic Differentiation of Stem Cells of Dental Apical Papilla (SCAPs). Biomaterials, 39, 145-154.
https://doi.org/10.1016/j.biomaterials.2014.11.007
|
[30]
|
Kamizaki, K., Endo, M., Minami, Y., et al. (2021) Role of Noncanonical Wnt Ligands and Ror-Family Receptor Tyrosine Kinases in the Development, Regeneration, and Diseases of the Musculoskeletal System. Developmental Dynamics, 250, 27-38. https://doi.org/10.1002/dvdy.151
|
[31]
|
Church, V., Nohno, T., Linker, C., et al. (2002) Wnt Regulation of Chon-drocyte Differentiation. Journal of Cell Science, 115, 4809-4818. https://doi.org/10.1242/jcs.00152
|
[32]
|
de Winter, T. and Nusse, R. (2021) Running against the Wnt: How Wnt/beta-Catenin Suppresses Adipogenesis. Frontiers in Cell and Developmental Biology, 9, Article ID: 627429. https://doi.org/10.3389/fcell.2021.627429
|
[33]
|
Liang, K., Du, Y., Chen, L., et al. (2020) Contrary Roles of Wnt/beta-Catenin Signaling in BMP9-Induced Osteogenic and Adipogenic Differentiation of 3T3-L1 Preadipocytes. Cell Biochemistry and Biophysics, 78, 347-356.
https://doi.org/10.1007/s12013-020-00935-0
|
[34]
|
Li, Y., Wu, B., Liang, J., et al. (2019) Isopsoralen Ameliorates H2O2-Induced Damage in Osteoblasts via Activating the Wnt/β-Catenin Pathway. Experimental and Therapeutic Medi-cine, 18, 1899-1906.
https://doi.org/10.3892/etm.2019.7741
|
[35]
|
Joeng, K.S., Lee, Y.C., Lim, J., et al. (2017) Osteocyte-Specific WNT1 Regulates Osteoblast Function during Bone Homeostasis. Journal of Clinical Investigation, 127, 2678-2688. https://doi.org/10.1172/JCI92617
|
[36]
|
Yang, Z., Liu, J., Fu, J., et al. (2022) Associations between WNT Signaling Pathway-Related Gene Polymorphisms and Risks of Osteoporosis Development in Chinese Postmenopausal Women: A Case-Control Study. Climacteric, 25, 257-263. https://doi.org/10.1080/13697137.2021.1941848
|