[1]
|
Lu, G., He, Q., Shen, Y. and Cao, F. (2018) Potential Biomarkers for Predicting Hemorrhagic Transformation of Ischemic Stroke. The International Journal of Neuroscience, 128, 79-89.
https://doi.org/10.1080/00207454.2017.1349766
|
[2]
|
Trouillas, P. and von Kummer, R. (2006) Classification and Pathogenesis of Cerebral Hemorrhages after Thrombolysis in Ischemic Stroke. Stroke, 37, 556-561. https://doi.org/10.1161/01.STR.0000196942.84707.71
|
[3]
|
Hacke, W., Kaste, M., Fieschi, C., von Kummer, R., Davalos, A., Meier, D., Larrue, V., Bluhmki, E., Davis, S., Donnan, G., Schneider, D., Diez-Tejedor, E. and Trouillas, P. (1998) Randomised Double-Blind Placebo-Controlled Trial of Thrombolytic Therapy with Intravenous Alteplase in Acute Ischaemic Stroke (ECASS II). Second European-Australasian Acute Stroke Study Investigators. The Lancet (London, England), 352, 1245-1251.
https://doi.org/10.1016/S0140-6736(98)08020-9
|
[4]
|
Cummins, P.M. (2012) Occludin: One Protein, Many Forms. Molecular and Cellular Biology, 32, 242-250.
https://doi.org/10.1128/MCB.06029-11
|
[5]
|
Akimoto, T., Takasawa, A., Murata, M., Kojima, Y., Takasawa, K., Nojima, M., Aoyama, T., Hiratsuka, Y., Ono, Y., Tanaka, S., Osanai, M., Hasegawa, T., Saito, T. and Sawada, N. (2016) Analysis of the Expression and Localization of Tight Junction Transmembrane Proteins, Claudin-1, -4, -7, Occludin and JAM-A, in Human Cervical Adenocarcinoma. Histology and Histopathology, 31, 921-931.
|
[6]
|
DeMaio, L., Rouhanizadeh, M., Reddy, S., Sevanian, A., Hwang, J. and Hsiai, T.K. (2006) Oxidized Phospholipids Mediate Occludin Expression and Phosphorylation in Vascular Endothelial Cells. American Journal of Physiology. Heart and Circulatory Physiology, 290, H674-H683. https://doi.org/10.1152/ajpheart.00554.2005
|
[7]
|
Yang, C., Hawkins, K.E., Doré, S. and Candelario-Jalil, E. (2019) Neuroinflammatory Mechanisms of Blood-Brain Barrier Damage in Ischemic Stroke. American Journal of Physiology. Cell Physiology, 316, C135-c153.
https://doi.org/10.1152/ajpcell.00136.2018
|
[8]
|
Strbian, D., Durukan, A., Pitkonen, M., Marinkovic, I., Tatlisumak, E., Pedrono, E., Abo-Ramadan, U. and Tatlisumak, T. (2008) The Blood-Brain Barrier Is Continuously Open for Several Weeks Following Transient Focal Cerebral Ischemia. Neuroscience, 153, 175-181. https://doi.org/10.1016/j.neuroscience.2008.02.012
|
[9]
|
Bernardo-Castro, S., Sousa, J.A., Brás, A., Cecília, C., Rodrigues, B., Almendra, L., Machado, C., Santo, G., Silva, F., Ferreira, L., Santana, I. and Sargento-Freitas, J. (2020) Pathophysiology of Blood-Brain Barrier Permeability Throughout the Different Stages of Ischemic Stroke and Its Implication on Hemorrhagic Transformation and Recovery. Frontiers in Neurology, 11, Article ID: 594672. https://doi.org/10.3389/fneur.2020.594672
|
[10]
|
Wang, X. and Lo, E.H. (2003) Triggers and Mediators of Hemorrhagic Transformation in Cerebral Ischemia. Molecular Neurobiology, 28, 229-244. https://doi.org/10.1385/MN:28:3:229
|
[11]
|
Jung, J.E., Kim, G.S., Chen, H., Maier, C.M., Narasimhan, P., Song, Y.S., Niizuma, K., Katsu, M., Okami, N., Yoshioka, H., Sakata, H., Goeders, C.E. and Chan, P.H. (2010) Reperfusion and Neurovascular Dysfunction in Stroke: From Basic Mechanisms to Potential Strategies for Neuroprotection. Molecular Neurobiology, 41, 172-179.
https://doi.org/10.1007/s12035-010-8102-z
|
[12]
|
Krueger, M., Bechmann, I., Immig, K., Reichenbach, A., Härtig, W. and Michalski, D. (2015) Blood-Brain Barrier Breakdown Involves Four Distinct Stages of Vascular Damage in Various Models Of Experimental Focal Cerebral Ischemia. Journal of Cerebral Blood Flow and Metabolism: Official Journal of the International Society of Cerebral Blood Flow and Metabolism, 35, 292-303. https://doi.org/10.1038/jcbfm.2014.199
|
[13]
|
Khatri, R., McKinney, A.M., Swenson, B. and Janardhan, V. (2012) Blood-Brain Barrier, Reperfusion Injury, and Hemorrhagic Transformation in Acute Ischemic Stroke. Neurology, 79, S52-S57.
https://doi.org/10.1212/WNL.0b013e3182697e70
|
[14]
|
Jiao, H., Wang, Z., Liu, Y., Wang, P. and Xue, Y. (2011) Specific Role of Tight Junction Proteins Claudin-5, Occludin, and ZO-1 of the Blood-Brain Barrier in a Focal Cerebral Ischemic Insult. Journal of Molecular Neuroscience: MN, 44, 130-139. https://doi.org/10.1007/s12031-011-9496-4
|
[15]
|
Yuan, S., Liu, K.J. and Qi, Z. (2020) Occludin Regulation of Blood-Brain Barrier and Potential Therapeutic Target in Ischemic Stroke. Brain Circulation, 6, 152-162. https://doi.org/10.4103/bc.bc_29_20
|
[16]
|
Kazmierski, R., Michalak, S., Wencel-Warot, A. and Nowinski, W.L. (2012) Serum Tight-Junction Proteins Predict Hemorrhagic Transformation in Ischemic Stroke Patients. Neurology, 79, 1677-1685.
https://doi.org/10.1212/WNL.0b013e31826e9a83
|
[17]
|
Koizumi, J., Kojima, T., Ogasawara, N., Kamekura, R., Kurose, M., Go, M., Harimaya, A., Murata, M., Osanai, M., Chiba, H., Himi, T. and Sawada, N. (2008) Protein Kinase C Enhances Tight Junction Barrier Function of Human Nasal Epithelial Cells in Primary Culture by Transcriptional Regulation. Molecular Pharmacology, 74, 432-442.
https://doi.org/10.1124/mol.107.043711
|
[18]
|
Mattiotti, A., Prakash, S., Barnett, P. and van den Hoff, M.J.B. (2018) Follistatin-Like 1 in Development and Human Diseases. Cellular and Molecular Life Sciences: CMLS, 75, 2339-2354. https://doi.org/10.1007/s00018-018-2805-0
|
[19]
|
Xi, Y., Hao, M., Liang, Q., Li, Y., Gong, D.W. and Tian, Z. (2020) Dynamic Resistance Exercise Increases Skeletal Muscle-Derived FSTL1 Inducing Cardiac Angiogenesis via DIP2A-Smad2/3 in Rats Following Myocardial Infarction. Journal of Sport and Health Science, 10, 594-603. https://doi.org/10.1016/j.jshs.2020.11.010
|
[20]
|
Liu, Y.P., Ju, M.L. and Yu, F.Q. (2020) Clinical Significance of FSTL 1, Bax, Bcl-2 in Acute Cerebral Infarction and Its Relationship with Hemorrhagic Transformation. European Review for Medical and Pharmacological Sciences, 24, 8447-8457.
|
[21]
|
Liang, X., Hu, Q., Li, B., McBride, D., Bian, H., Spagnoli, P., Chen, D., Tang, J. and Zhang, J.H. (2014) Follistatin-Like 1 Attenuates Apoptosis via Disco-Interacting Protein 2 Homolog A/Akt Pathway after Middle Cerebral Artery Occlusion in Rats. Stroke, 45, 3048-3054. https://doi.org/10.1161/STROKEAHA.114.006092
|
[22]
|
Xiang, S., Zhang, Y., Jiang, T., Ke, Z., Shang, Y., Ning, W., Yang, Z. and Zhang, T. (2020) Knockdown of Follistatin-Like 1 Disrupts Synaptic Transmission in Hippocampus and Leads to Cognitive Impairments. Experimental Neurology, 333, Article ID: 113412. https://doi.org/10.1016/j.expneurol.2020.113412
|
[23]
|
Milanović, Z., Sporiš, G. and Weston, M. (2015) Effectiveness of High-Intensity Interval Training (HIT) and Continuous Endurance Training for VO2max Improvements: A Systematic Review and Meta-Analysis of Controlled Trials. Sports Medicine (Auckland, N.Z.), 45, 1469-1481. https://doi.org/10.1007/s40279-015-0365-0
|
[24]
|
Ruan, Y., Qiu, X., Lv, Y.D., Dong, D., Wu, X.J., Zhu, J. and Zheng, X.Y. (2019) Kainic Acid Induces Production and Aggregation of Amyloid β-Protein and Memory Deficits by Activating Inflammasomes in NLRP3- and NF-κB-Stimulated Pathways. Aging, 11, 3795-3810. https://doi.org/10.18632/aging.102017
|
[25]
|
Kwon, O.H., Cho, Y.Y., Kim, T.W. and Chung, S. (2019) O-GlcNAcylation of Amyloid-β Protein Precursor by Insulin Signaling Reduces Amyloid-β Production. Journal of Alzheimer’s Disease: JAD, 69, 1195-1211.
https://doi.org/10.3233/JAD-190060
|