[1]
|
Li, L., Bao, J., Chang, Y., et al. (2021) Gut Microbiota May Mediate the Influence of Periodontitis on Prediabetes. Jour-nal of Dental Research, 100, 1387-1396. https://doi.org/10.1177/00220345211009449
|
[2]
|
Shajib, M.S. and Khan, W.I. (2015) The Role of Serotonin and Its Receptors in Activation of Immune Responses and Inflammation. Acta Physi-ologica (Oxford), 213, 561-574. https://doi.org/10.1111/apha.12430
|
[3]
|
Liu, B.N., Liu, X.T., Liang, Z.H., et al. (2021) Gut Microbiota in Obesity. World Journal of Gastroenterology, 27, 3837-3850. https://doi.org/10.3748/wjg.v27.i25.3837
|
[4]
|
Scheithauer, T., Rampanelli, E., Nieuwdorp, M., et al. (2020) Gut Microbiota as a Trigger for Metabolic Inflammation in Obesity and Type 2 Diabetes. Frontiers in Immunology, 11, Arti-cle ID: 571731.
https://doi.org/10.3389/fimmu.2020.571731
|
[5]
|
李雷, 张帆, 赵进和, 等. 老年2型糖尿病患者相关炎症因子及肠道菌群多样性变化研究[C]//浙江省医学会中毒学分会. 2019年浙江省医学会中毒学学术大会论文汇编. 湖州: 湖州市第三人民医院老年病科, 2019: 7.
https://doi.org/10.26914/c.cnkihy.2019.102476
|
[6]
|
Patil, R. and Arvindekar, A. (2021) Glycation of Gut Proteins Initiates Microbial Dysbiosis and Can Promote Establishment of Diabetes in Experimental Animals. Microbial Patho-genesis, 152, Article ID: 104589.
https://doi.org/10.1016/j.micpath.2020.104589
|
[7]
|
Zhou, Z., Sun, B., Yu, D., et al. (2022) Gut Microbiota: An Important Player in Type 2 Diabetes Mellitus. Frontiers in Cellular and Infection Microbiology, 12, Article ID: 834485. https://doi.org/10.3389/fcimb.2022.834485
|
[8]
|
Saito, T., Hayashida, H. and Furugen, R. (2007) Comment on: Cani et al. (2007) Metabolic Endotoxemia Initiates Obesity and Insulin Resistance: Diabetes 56: 1761-1772. Diabetes, 56, e20. https://doi.org/10.2337/db07-1181
|
[9]
|
He, F.F. and Li, Y.M. (2020) Role of Gut Microbiota in the Devel-opment of Insulin Resistance and the Mechanism Underlying Polycystic Ovary Syndrome: A Review. Journal of Ovari-an Research, 13, Article No. 73.
https://doi.org/10.1186/s13048-020-00670-3
|
[10]
|
Al-Ishaq, R.K., Samuel, S.M. and Büsselberg, D. (2023) The In-fluence of Gut Microbial Species on Diabetes Mellitus. International Journal of Molecular Sciences, 24, Article No. 8118. https://doi.org/10.3390/ijms24098118
|
[11]
|
Iatcu, C.O., Steen, A. and Covasa, M. (2021) Gut Microbiota and Complications of Type-2 Diabetes. Nutrients, 14, Article No. 166. https://doi.org/10.3390/nu14010166
|
[12]
|
McGlone, E.R. and Bloom, S.R. (2019) Bile Acids and the Metabolic Syndrome. Annals of Clinical Biochemistry, 56, 326-337. https://doi.org/10.1177/0004563218817798
|
[13]
|
Sonne, D.P., van Nierop, F.S., Kulik, W., et al. (2016) Postprandial Plasma Concentrations of Individual Bile Acids and FGF-19 in Patients with Type 2 Diabetes. The Journal of Clinical Endocrinology & Metabolism, 101, 3002-3009.
https://doi.org/10.1210/jc.2016-1607
|
[14]
|
Wang, S., Deng, Y., Xie, X., et al. (2018) Plasma Bile Acid Changes in Type 2 Diabetes Correlated with Insulin Secretion in Two-Step Hyperglycemic Clamp. Journal of Diabetes, 10, 874-885. https://doi.org/10.1111/1753-0407.12771
|
[15]
|
Versteeg, B., Himschoot, M., van den Broek, I.V., et al. (2015) Urogenital Chlamydia trachomatis Strain Types, Defined by High-Resolution Multilocus Sequence Typing, in Relation to Ethnicity and Urogenital Symptoms among a Young Screening Population in Amsterdam, The Netherlands. Sexually Transmitted Infections, 91, 415-422.
https://doi.org/10.1136/sextrans-2014-051790
|
[16]
|
Sachdev, S., Wang, Q., Billington, C., et al. (2016) FGF 19 and Bile Acids Increase Following Roux-en-Y Gastric Bypass but Not After Medical Management in Patients with Type 2 Diabetes. Obesity Surgery, 26, 957-965.
|
[17]
|
Wu, Y., Zhou, A., Tang, L., et al. (2020) Bile Acids: Key Regulators and Novel Treatment Targets for Type 2 Diabetes. Journal of Diabetes Research, 2020, Article ID: 6138438. https://doi.org/10.1155/2020/6138438
|
[18]
|
Katafuchi, T. and Makishima, M. (2022) Molecular Basis of Bile Ac-id-FXR-FGF15/19 Signaling Axis. International Journal of Molecular Sciences, 23, Article No. 6046. https://doi.org/10.3390/ijms23116046
|
[19]
|
Chávez-Talavera, O., Wargny, M., Pichelin, M., et al. (2020) Bile Acids Associate with Glucose Metabolism, but Do Not Predict Conversion from Impaired Fasting Glucose to Diabetes. Metab-olism, 103, Article ID: 154042.
https://doi.org/10.1016/j.metabol.2019.154042
|
[20]
|
Lu, J., Wang, S., Li, M., et al. (2021) Association of Serum Bile Acids Profile and Pathway Dysregulation with the Risk of Developing Diabetes among Normoglycemic Chinese Adults: Findings from the 4C Study. Diabetes Care, 44, 499-510. https://doi.org/10.2337/dc20-0884
|
[21]
|
Sayin, S.I., Wahlström, A., Felin, J., et al. (2013) Gut Microbiota Regulates Bile Acid Metabolism by Reducing the Levels of Tauro-Beta-Muricholic Acid, a Naturally Occurring FXR Antagonist. Cell Metabolism, 17, 225-235.
https://doi.org/10.1016/j.cmet.2013.01.003
|
[22]
|
Moore, R.H., Chothe, P. and Swaan, P.W. (2013) Transmembrane Domain V Plays a Stabilizing Role in the Function of Human Bile Acid Transporter SLC10A2. Biochemistry, 52, 5117-5124. https://doi.org/10.1021/bi400028q
|
[23]
|
Hu, X., Bonde, Y., Eggertsen, G., et al. (2014) Muricholic Bile Acids Are Potent Regulators of Bile Acid Synthesis via a Positive Feedback Mechanism. Journal of Internal Medicine, 275, 27-38. https://doi.org/10.1111/joim.12140
|
[24]
|
Sorribas, M., Jakob, M.O., Yilmaz, B., et al. (2019) FXR Modulates the Gut-Vascular Barrier by Regulating the Entry Sites for Bacterial Translocation in Experimental Cirrhosis. Journal of Hepatology, 71, 1126-1140.
https://doi.org/10.1016/j.jhep.2019.06.017
|
[25]
|
Zhang, Y., Hagedorn, C.H. and Wang, L. (2011) Role of Nuclear Receptor SHP in Metabolism and Cancer. Biochimica et Biophysica Acta, 1812, 893-908. https://doi.org/10.1016/j.bbadis.2010.10.006
|
[26]
|
Ma, K., Saha, P.K., Chan, L., et al. (2006) Farnesoid X Receptor Is Essential for Normal Glucose Homeostasis. Journal of Clinical Investigation, 116, 1102-1109. https://doi.org/10.1172/JCI25604
|
[27]
|
Marcelin, G., Jo, Y.-H., Li, X.S., et al. (2013) Central Action of FGF19 Reduces Hypothalamic AGRP/NPY Neuron Activity and Improves Glucose Metabolism. Molecular Metabolism, 3, 19-28.
|
[28]
|
Zhou, M., Luo, J., Chen, M., et al. (2017) Mouse Species-Specific Control of Hepatocarcinogenesis and Metabolism by FGF19/FGF15. Journal of Hepatology, 66, 1182-1192. https://doi.org/10.1016/j.jhep.2017.01.027
|
[29]
|
Wahlström, A., Sayin, S.I., Marschall, H.U., et al. (2016) Intestinal Crosstalk between Bile Acids and Microbiota and Its Impact on Host Metabolism. Cell Metabolism, 24, 41-50. https://doi.org/10.1016/j.cmet.2016.05.005
|
[30]
|
Sacks, D., Baxter, B., Campbell, B., et al. (2018) Multisociety Consensus Quality Improvement Revised Consensus Statement for Endovascular Therapy of Acute Ischemic Stroke. In-ternational Journal of Stroke, 13, 612-632.
https://doi.org/10.1016/j.jvir.2017.11.026
|
[31]
|
邹步, 唐莹, 杨文玲, 等. 肠道菌群-FXR轴在代谢性疾病中的作用[J]. 中国病理生理杂志, 2019, 35(9): 1716-1720.
|
[32]
|
龚彤, 陈国芳, 刘超. 肠道菌群——胆汁酸通路对代谢性疾病的影响[J]. 中国糖尿病杂志, 2019, 27(12): 953-955.
|
[33]
|
Suh, J.M., Jonker, J.W., Ahmadian, M., et al. (2014) Endocrinization of FGF1 Produces a Neomorphic and Potent Insulin Sensitizer. Nature, 513, 436-439. https://doi.org/10.1038/nature13540
|
[34]
|
肖丹, 邵勇. 胆汁酸膜受体TGR5与代谢相关疾病的研究进展[J]. 肝脏, 2013, 18(11): 776-779.
|
[35]
|
Pathak, P., Xie, C., Nichols, R.G., et al. (2018) Intestine Farnesoid X Receptor Agonist and the Gut Microbiota Activate G-Protein Bile Acid Receptor-1 Signaling to Improve Metabolism. Hepatology, 68, 1574-1588.
https://doi.org/10.1002/hep.29857
|
[36]
|
Tomaro-Duchesneau, C., LeValley, S.L., Roeth, D., et al. (2020) Discovery of a Bacterial Peptide as a Modulator of GLP-1 and Metabolic Disease. Scientific Reports, 10, Article No. 4922. https://doi.org/10.1038/s41598-020-61112-0
|
[37]
|
Deng, L., Yang, Y. and Xu, G. (2022) Empagliflozin Amelio-rates Type 2 Diabetes Mellitus-Related Diabetic Nephropathy via Altering the Gut Microbiota. Biochimica et Biophysica Acta (BBA)—Molecular and Cell Biology of Lipids, 1867, Article ID: 159234. https://doi.org/10.1016/j.bbalip.2022.159234
|
[38]
|
Huang, S., Ma, S., Ning, M., et al. (2019) TGR5 Agonist Ame-liorates Insulin Resistance in the Skeletal Muscles and Improves Glucose Homeostasis in Diabetic Mice. Metabolism, 99, 45-56.
https://doi.org/10.1016/j.metabol.2019.07.003
|
[39]
|
Chen, B., Bai, Y., Tong, F., et al. (2023) Glycoursodeoxycholic Acid Regulates Bile Acids Level and Alters Gut Microbiota and Glycolipid Metabolism to Attenuate Diabetes. Gut Mi-crobes, 15, Article ID: 2192155.
https://doi.org/10.1080/19490976.2023.2192155
|
[40]
|
Qiu, P., Ishimoto, T., Fu, L., et al. (2022) The Gut Microbi-ota in Inflammatory Bowel Disease. Frontiers in Cellular and Infection Microbiology, 12, Article ID: 733992. https://doi.org/10.3389/fcimb.2022.733992
|
[41]
|
Vettorazzi, J.F., Ribeiro, R.A., Borck, P.C., et al. (2016) The Bile Acid TUDCA Increases Glucose-Induced Insulin Secretion via the cAMP/PKA Pathway in Pancreatic Beta Cells. Me-tabolism, 65, 54-63.
https://doi.org/10.1016/j.metabol.2015.10.021
|
[42]
|
Castellanos-Jankiewicz, A., Guzmán-Quevedo, O., Fénelon, V.S., et al. (2021) Hypothalamic Bile Acid-TGR5 Signaling Protects from Obesity. Cell Metabolism, 33, 1483-1492.e10. https://doi.org/10.1016/j.cmet.2021.04.009
|
[43]
|
Mantovani, A., Dalbeni, A., Peserico, D., et al. (2021) Plasma Bile Acid Profile in Patients with and without Type 2 Diabetes. Metabolites, 11, Article No. 453. https://doi.org/10.3390/metabo11070453
|
[44]
|
Zhang, L., Wu, W., Lee, Y.K., et al. (2018) Spatial Heterogeneity and Co-Occurrence of Mucosal and Luminal Microbiome across Swine Intestinal Tract. Frontiers in Microbiology, 9, Article No. 48.
https://doi.org/10.3389/fmicb.2018.00048
|
[45]
|
Ðanić, M., Stanimirov, B., Pavlović, N., et al. (2018) Pharmaco-logical Applications of Bile Acids and Their Derivatives in the Treatment of Metabolic Syndrome. Frontiers in Pharma-cology, 9, Article No. 1382.
https://doi.org/10.3389/fphar.2018.01382
|